ASN's Mission

To create a world without kidney diseases, the ASN Alliance for Kidney Health elevates care by educating and informing, driving breakthroughs and innovation, and advocating for policies that create transformative changes in kidney medicine throughout the world.

learn more

Contact ASN

1401 H St, NW, Ste 900, Washington, DC 20005

email@asn-online.org

202-640-4660

The Latest on X

Kidney Week

Abstract: SA-PO800

Pegcetacoplan for Post-transplant Recurrent C3 Glomerulopathy (C3G) or Immune Complex Membranoproliferative Glomerulonephritis (IC-MPGN) in NOBLE: 52-Week Patient Evolution

Session Information

Category: Glomerular Diseases

  • 1402 Glomerular Diseases: Clinical, Outcomes, and Therapeutics

Authors

  • Java, Anuja, Washington University in St Louis School of Medicine, St Louis, Missouri, United States
  • Bomback, Andrew S., Columbia University Irving Medical Center, New York, New York, United States
  • Kavanagh, David, Newcastle University, Newcastle upon Tyne, Tyne and Wear, United Kingdom
  • Remuzzi, Giuseppe, Istituto di Ricerche Farmacologiche Mario Negri, Bergamo, Italy
  • Sunder-Plassmann, Gere, Medizinische Universitat Wien, Vienna, Austria
  • Kanellis, John, Monash Medical Centre, Clayton, Victoria, Australia
  • Daina, Erica, Istituto di Ricerche Farmacologiche Mario Negri, Bergamo, Italy
  • Walker, Patrick D., Arkana Laboratories, Little Rock, Arkansas, United States
  • Wang, Zhongshen, Apellis Pharmaceuticals Inc, Waltham, Massachusetts, United States
  • Ahmad, Zurish, Apellis Pharmaceuticals Inc, Waltham, Massachusetts, United States
  • Fakhouri, Fadi, Centre Hospitalier Universitaire Vaudois, Lausanne, Vaud, Switzerland
Background

Pegcetacoplan (Peg; targeted C3/C3b inhibitor) may prevent kidney damage in C3G and primary IC-MPGN. NOBLE (NCT04572854) randomized 13 patients with post-transplant recurrent C3G or primary IC-MPGN. At week 12, Peg was well tolerated and showed improvements in histologic, clinical, and biomarker parameters of disease.

Methods

We conducted a post-hoc analysis of 9 NOBLE patients treated with subcutaneous Peg 1080 mg twice weekly plus standard of care for 52 weeks; of these, treatment completers had >80% compliance (n=7).

Results

At week 52, 5/9 (55.6%) patients had reduced C3c staining (p=0.0423 vs baseline). 7/9 (77.8%) had decreased activity score compared to baseline and 2/8 (25%) had absent electron microscopy deposits at week-52 biopsy. Patients with >1 g/g proteinuria at baseline had a median 56.4% decrease in proteinuria (Table). 7/9 (77.8%) patients had stable/improved estimated glomerular filtration rate, and 8/9 (88.9%) had both increased serum C3 and decreased sC5b-9. No meningitis cases, graft losses, or deaths were reported. Non-serious rejection episodes were reported for 2/9 (22.2%) patients. 1 patient (11.1%) discontinued for serious adverse event of weight loss.

Conclusion

By inhibiting the breakdown of C3, Peg achieved meaningful histology improvements for 8/9 (88.9%) patients while also improving disease parameters, increasing serum C3, and decreasing plasma sC5b-9. The Phase 3 VALIANT (NCT05067127) trial will further evaluate the safety and efficacy of Peg in patients with native kidney or post-transplant C3G or primary IC-MPGN.